Untangling complex syste.., p.40

Untangling Complex Systems, page 40

 

Untangling Complex Systems
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  • How does glycolysis originate oscillations?

  • Describe the signal transduction model without feedback.

  • Describe the signal transduction model with linear feedback

  • Describe the signal transduction model with linear feedback and Michaelis-Menten

  de-activation reaction.

  • Describe the signal transduction model with ultrasensitive feedback.

  • What is an epigenetic event, and which are the protagonists?

  • Which are the favorable conditions to observe oscillations in an epigenetic event?

  • Make examples of biological rhythms.

  192

  Untangling Complex Systems

  • Explain the mechanism of magnitude amplification.

  • Which are the mechanisms for having sensitivity amplification?

  • How do we adapt to stimuli?

  7.8 KEY WORDS

  Proteins; Metabolism; Signal Transduction Pathway; Ultrasensitivity; Hysteresis; Epigenesis;

  Homeostasis; Ultradian, circadian and infradian rhythms; Amplification in sensing; Adaptation in

  sensing.

  7.9 HINTS FOR FURTHER READING

  • Walsh et al. (2018) pinpoint eight compounds as essential ingredients of metabolism within

  a cell. Seven of them use group transfer chemistry to drive otherwise unfavorable biosyn-

  thetic equilibria. They are ATP (for phosphoryl transfers), acetyl-CoA and carbamoyl phos-

  phate (for acyl transfers), S-adenosylmethionine (for methyl transfers), Δ2-isopentenyl-PP

  (for prenyl transfers), UDP-glucose (for glucosyl transfers), and NAD(P)H/NAD(P)+ (for

  electron and ADP-ribosyl transfers). The eighth key metabolite is O .

  2

  • The possible mechanisms for generating ultra-sensitivity can be learned by reading the

  series of three papers by Ferrell and Ha (2014a–c).

  • If you want to deepen the subject of biochemical oscillators and cellular rhythms, you

  can read Goldbeter (1996); Goldbeter and Caplan (1976); Ferrell et al. (2011); Glass and

  Mackey (1988).

  • A delightful book, about the action of the human brain on physiological processes, such as

  hunger, thirst, sex, and sleep, is that by Young (2012).

  7.10 EXERCISES

  7.1. Compare mechanism A, B, and C of Figure 7.26 and determine which one admits tr(J) = 0

  as possible solution.

  7.2. Consider the simplest model of a signaling event (Figure 7.10). Determine its response curve, i.e., how (χ * )

  [ ] / − [ ]

  R

  ss changes with the ratio k 1 S

  k 1 P .

  7.3. Regarding the model of signaling process with linear feedback depicted in Figure 7.12, determine the expression of the maximum for the forward rate. In case the term k [

  1 S] is

  negligible, which is the expression of the response curve?

  7.4. Build the rate balance plot and the stimulus-response curve for the signaling system with

  linear feedback and a back reaction that becomes saturated (based on the Michaelis-

  Menten mechanism). Such system is described by the mechanism of Figure 7.14. Assume

  that the values of the parameters appearing in equation [7.23] are: k [ R]

  /

  f

  tot = 0.7 t−1; ( kh 2 CP

  [ R] )

  /[ R]

  tot = 0.16 t−1; KM 2

  tot = 0.029. Plot the curves of the forward reaction for the following

  values of the term k [

  1 S]: 0; 0.04; 0.073; 0.093.

  A

  B

  C

  k 1

  k 1

  k 1

  X

  X

  X

  k 2

  k 2

  k 2

  X + Y

  2 Y

  X + Y

  Z

  X

  Y

  k 3

  k 3

  k 3

  Y

  Y

  Y

  FIGURE 7.26 Three distinct mechanisms.

  The Emergence of Temporal Order within a Living Being

  193

  7.5. Determine the properties of the stationary state for the system of two differential equations

  [7.27] describing how the [mRNA] and [ Y] change over time. Assume, for simplicity, that

  there is only one TF, its association coefficient is ni = 1 and its concentration [ TF] is fixed.

  7.11 SOLUTIONS TO THE EXERCISES

  7.1. Regarding mechanism A, the differential equations regarding X and Y are:

  d [ X ] = v 1 − k 2[ X] Y[]

  dt

  d Y

  [ ] = k 2[ X] Y[]− k 3 Y[]

  dt

  The terms of the Jacobian are

  X

  2 [

  ]

  2 [ ]

  2 [

  ]

  2 [ ]

  x = − k

  Y ; X y = − k X ; Yx = k Y ; Yy = k X

  − k

  ss

  ss

  ss

  ss

  3.

  The determinant det( J ) = k 3 k 2[ X ] ss > 0.

  The trace is tr( J ) = k 2 ([ X

  )

  ss

  ] − Y

  [ ss

  ]

  − k 3 can be positive, negative or null.

  Regarding mechanism B, the differential equations are

  d [ X ] = v 1 − k 2[ X] Y[]

  dt

  d Y

  [ ] = − k 2[ X] Y[]− k 3 Y[]

  dt

  Therefore, the terms of the Jacobian are

  X x = − k 2 Y

  [ s] s; X y = − k 2[ X s] s; Yx = − k 2 Y

  [ s] s; Yy = − k 2[ X s] s − k 3.

  The determinant det( J ) = k 3 k 2[ X ] ss > 0.

  The trace is tr( J ) = − k 2 ([ X

  )

  ss

  ] + Y

  [ ss

  ]

  − k 3 can be only negative.

  Regarding mechanism C, the differential equations are

  d [ X ] = v 1 − k 2[ X]

  dt

  d Y

  [ ] = k 2[ X]− k 3 Y[]

  dt

  The terms of the Jacobian are

  X x = − k 2; X y = 0; Yx = k 2; Yy = − k 3.

  The determinant det( J ) = k 3 k 2 > 0.

  The trace is tr( J ) = − k 2 − k 3 can be only negative.

  Only mechanism A, having an autocatalytic step, can give a null trace of Jacobian and

  hence a limit cycle as a possible stable solution.

  7.2. The response curve describes the dependence of (χ *)

  R ss on the ratio k 1[ S ] k

  / 1

  − [ P]. To obtain

  such a relation, we impose equation [7.19] equal to zero. After a few simple mathematical

  steps, we obtain

  1

  (χ ) =

  R* ss

  k−1[ P]

  1+ k 1[ S]

  194

  Untangling Complex Systems

  1.0

  0.8

  0.6

  ) SS∗

  ( χ R 0.4

  0.2

  0.0 0 1 2 3 4 5 6 7 8 9 10

  k 1[ S]/ k–1[ P]

  FIGURE 7.27 Response curve for the most straightforward signaling system.

  This equation is a hyperbolic function. It is shown in Figure 7.27. Note that when the

  ratio k

  χ

  1[ S ] k

  / 1

  − [ P] is 1, (

  * )

  R ss is equal to 0.5, that is 50% of [ R] is in its activated state.

  tot

  Moreover, (χ *)

  R ss goes asymptotically to 1.

  7.3. The equation relative to the forward rate ( v ) for a signaling system with linear feedback is

  for

  v

  2

  1 [ ]

  χ * ( [ ]

  1 [ ])

  [ ]

  for = k

  S +

  k R

  − k S − k R

  χ

  R

  f

  tot

  f

  tot

  R*

  To find its maximum as a function of χ R*, we calculate the first derivative of vfor with

  respect to χ R*. We obtain

  dv for = ( k [ ] 1[ ]) 2 [ ]

  f R

  − k S − k R

  χ

  tot

  f

  tot

  R*

  dχ R*

  ( k [ ] 1[ ])

  f R

  − k S

  It is equal to zero when χ

  tot

  =

  .

  R*

  2 k [ ]

  f R tot

  In

  case

  k [ R]

  [ S], χ ≈ 0 5.

  f

  tot >> k 1

  R*

  .

  When

  k [

  1 S] is negligible when compared to the other terms of equation [7.22], the over-

  all rate of R* formation becomes:

  dχ

  R* ≈ χ ( [ ] ) − [ ]

  2 − −1[ ]

  *

  k R

  k R

  χ * k P χ

  dt

  R

  f

  tot

  f

  tot

  R

  R*

  In such case, the equation of the response curve is

  k−1[ P]

  χ ≈1−

  R*

  k [ ]

  f R tot

  7.4. In the case of a signaling system with a linear feedback and a back reaction with satura-

  tion (based on a Michaelis-Menten mechanism), a continuous growth of the stimulus from

  zero value, shifts the system from a stable fixed point to a new stable one in an irreversible

  The Emergence of Temporal Order within a Living Being

  195

  0.8

  0.7

  0.6

  0.16

  0.5

  χR∗ 0.4

  te

  0.3

  0.08

  Ra

  0.2

  0.1

  0.0

  0.00

  0.0

  0.2

  0.4

  0.6

  0.8

  1.0

  0.00

  0.02

  0.04

  0.06

  0.08

  0.10

  (a)

  χR∗

  (b)

  k 1[ S]

  FIGURE 7.28 Rate balance plot (a) and stimulus-response curve (b) for a signaling system with linear feed-

  back and a Michaelis-Menten back reaction. In both (a) and (b), the stable and unstable fixed points are indi-

  cated by squares and circles, respectively.

  manner (This behavior is also exhibited in the case of a signaling system with sigmoidal

  positive feedback. Read the next part of paragraph 7.3.2). The rate balance plot and the

  stimulus-response curve built through the values of the exercise are plotted in Figure 7.28.

  7.5. Based on the assumptions of the exercise, the differential equations [7.27] become:

  d [mRNA]

  

  TF

  [ ] 

  = ktr 

   k mRNA

  0

  dt

   TF

   − [

  ] =

   [

  ]+ K

  d

  

  d Y

  [ ]

  

  [ Y ] 

  = k [mRNA]− khET 

   0

  dt

  tl

   KM +

   =

  

  [ Y ]

  The Jacobian for the system is:

   − kd

  0

  

  

  

  J =

  khET K

   k

  M

  

  tl

  −

  

  2 

  

  ( K [ ] )

  M + Y

  ss

  

  The trace and the determinant of J are:

  khET K

  tr ( J ) = − k

  M

  d − (

  0

  + [ ] ) <

  2

  KM

  Y ss

  kdkhET K

  det ( J

  M

  ) = (

  0

  + [ ] ) >

  2

  KM

  Y ss

  

  2

  2

  khET K

  The discriminant ∆ = ( tr( J )) − 4 det ( J ) 

  M

  

  kd

  > 0

  

   =

  −

  

  2 

  KM

  Y

  

  

  ( +[ ] ss) 

  These conditions correspond to a steady-state solution that is a stable node.

  The Emergence of

  8 Temporal Order in a

  Chemical Laboratory

  Experiment is the interrogation of Nature.

  Robert Boyle (1627–1691 AD)

  8.1 INTRODUCTION

  Oscillations are everywhere in nature: in the Universe, in our Solar System, in our planet, in ecosystems,

  in the economy, and inside every living being, as we have learned in the previous chapters. It was

  a great surprise to observe oscillations also in chemical laboratories because chemical oscillations

  were imagined as a sort of perpetual motion machines in a beaker, in sharp contradiction with the

  Second Law of Thermodynamics.

  8.2 THE DISCOVERY OF OSCILLATING CHEMICAL REACTIONS

  The first report of chemical oscillations in a lab was made by Robert Boyle in the late seventeenth

  century. At that time, Boyle was investigating the luminescence of phosphorus in the gaseous phase,

  in liquid solutions, and in the solid phase (Harvey 1957). He found with wonder that the oxidation

  of phosphorous produces flashes of light. The second report didn’t occur until the beginning of the

  nineteenth century when A. T. Fechner (1828) described an electrochemical cell producing an oscil-

  lating current. About seventy years later, in the late 1890s, J. Liesegang (1896) discovered periodic

  precipitation patterns in space and time (first mentioned by F. F. Runge in 1855), and Ostwald (1899)

  observed that the rate of chromium dissolution in acid periodically increased and decreased. But all

  these experiments, involving heterogeneous systems, did not undermine the widespread idea that

  a chemical reaction proceeding towards its equilibrium state cannot show oscillations.

  During the early decades of the twentieth century, Lotka (1910, 1920a, 1920b) published a hand-

  ful of theoretical papers on chemical oscillations. His models inspired ecologists. The most famous

  one is named as the Lotka-Volterra model, which is used to characterize the predator-prey interac-

  tion in an ecosystem as we have seen in Chapter 5.

  The report of the first homogeneous isothermal chemical oscillator is ascribed to Bray (1921),

  and Bray and Liebhafsky (1931), who studied the catalytic decomposition of hydrogen peroxide by

  the iodic acid-iodine couple. The mixture consisted of H O , KIO , and H SO and the reactions

  2

  2

  3

  2

  4

  were:

  5H2O2 + I2 = 2HIO3 + 4H2O [8.1]

  5H2O2 + 2HIO3 = 5O2 + I2 + 6H2O [8.2]

  where hydrogen peroxide plays as both an oxidizing and reducing agent.

  197

  198

  Untangling Complex Systems

  Bray serendipitously observed that the concentration of iodine and the rate of oxygen evolu-

  tion vary almost periodically. He offered no explanation for the oscillation other than referring to

  Lotka’s theoretical work. He suggested that autocatalysis was involved in the mechanism of its reac-

  tion. Bray’s results triggered more works devoted to debunking them than to explaining it mecha-

  nistically (Nicolis and Portnow 1973). Most chemists tried to prove that the cause of the oscillations

  was some unknown heterogeneous impurity.

  The next experimental observation of an oscillating reaction was made again accidentally by the

  Russian biophysicist Boris Belousov. During the Second World War, he was working on projects in

  radiation and chemical warfare protection. By investigating methods for removing toxic agents from

  the human body, Belousov developed a keen interest in the role of biochemical metabolic processes

  (Kiprijanov 2016). After the war, part of the Belousov’s research included the Krebs cycle. He was

  looking for an inorganic analog of the citric acid cycle.1 In 1950, he studied the reaction of citric acid with bromate and ceric ions (Ce+4) in sulfuric acid. He expected to see the monotonic depletion of the yellow ceric ions into the colorless cerous (Ce+3) ions. Instead, he astonishingly observed

  that the color of the solution repeatedly oscillated with a period of one minute or so between pale

  yellow and colorless. Belousov also noted that if the same reaction was carried out unstirred in

  a graduated cylinder, the solution exhibited awesome traveling waves of yellow. Belousov care-

  fully characterized the phenomenon and submitted a manuscript containing the recipe in 1951 to

  the Soviet periodical Journal of General Chemistry. Unfortunately, his paper was quickly rejected

  because the phenomenon described by Belousov was in contradiction with the Second Law of

  Thermodynamics. Belousov attempted a second submission to the Russian Kinetics and Catalysis

  in 1955. But his manuscript was rejected again for the same reason—phenomenon like that could

  not occur. Although Belousov furnished a recipe and photographs of the different stages of the

  oscillations, the evidence was judged insufficient. Therefore, he decided to work more on that reac-

 

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